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doi: 10.1016/j.freeradbiomed.2020.02.025 68

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These results supported that compound 102 was a specific GPX4 activator, and increasing GPX4 activity would be an alternative way to suppress ferroptosis

A: There's no strong human evidence that it directly "burns fat" through thermogenesis or lipolysis pathways, the way true fat-loss drugs aim to do

This would be an astonishing discovery in the history of science

doi:10.1074/jbc.R112.356485 Xiao B, Sanders MJ, Underwood E, Heath R, Mayer FV, Carmena D, Jing C, Walker PA, Eccleston JF, Haire LF, Saiu P, Howell SA, Aasland R, Martin SR, Carling D, Gamblin SJ (2011) Structure of mammalian AMPK and its regulation by ADP