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Furthermore, in a mouse model of intranasal infection, the sodA mutant displayed a slower multiplication rate in the lungs within the first 24 h and a delayed appearance in the bloodstream, indicating the contribution of SodA to the virulence of pneumococcal infections (Yesilkaya et al., 2000)
10.1111/ane.13293 23
DOI: 10.1002/biof.5520170101
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