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FIGURE 2 Furthermore, neonates have larger sized RBCs, and these cells have a shorter lifespan compared to that of the adult (120 days)

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Together, these interconnected signaling cascades reshape transcriptional programs in fibroblasts, AECs, and endothelial cells, promoting myofibroblast differentiation, epithelial cell dysfunction, immune dysregulation, and excessive ECM deposition, which collectively drive the onset and progression of pulmonary fibrosis [33, 39] (Fig

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