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The neurological aspects of the disease are thought to arise from the accumulation of methylmalonyl- CoA due to the requirement of B 12 as a cofactor to the enzyme methylmalonyl-CoA mutase
The only practical difference is dose range
BPC-157 is studied in preclinical research on tendon and ligament healing, muscle injury models, gastric ulcer protection, intestinal barrier function, and vascular endothelial growth signaling
It promotes the formation of new capillaries in damaged tissue which is critical for healing because repair requires oxygen and nutrient delivery
Its way bigger physically then the L.C
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