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In triple-negative breast cancer (TNBC) cells treated with isorhamnetin (IH) and chloroquine (CQ), ROS induced Drp1-dependent mitochondrial fission and apoptosis by mediating the activation and mitochondrial translocation of CaMKII

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IV administration avoids GI irritation common with oral zinc

At this point, GPX1 surprisingly appears to enhance the initial inflammatory response by initiating the production of pro-inflammatory cytokines to enhance the LPS-induced inflammatory response (Lubos et al., 2010)