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Defects in this gene result in familial adenomatous polyposis (FAP), an autosomal dominant precancerous condition that often progresses to malignancy

Since there's no general right answer, it's important to find the one that's right for you

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some report distinct dysfunction in 5FU-resistant models, and some, like our organoid model presented in Extended Data Fig

Pharmacological upregulation of Hrd1 expression or activity may accelerate the clearance of misfolded proteins, thereby reducing ER stress and preventing cardiomyocyte apoptosis, an approach particularly relevant in diabetic cardiomyopathy