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GLP-1 activation can reduce the pathological process of amyloid- plaque aggregation/deposition and tau hyperphosphorylation enhanced by neuroinflammation, thus preventing age-related neurodegenerative changes, such as decline of learning and memory (Li et al., 2012)

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Prolonged receptor stimulation provides reproducible outcomes and allows the study of long-term cumulative effects

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The treatment-policy estimand is closer to intention-to-treat: it estimates the effect regardless of whether participants stayed on treatment or used rescue therapies