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Bioavailability is usually determined by calculating the respective plasma drug exposure assessed as the total area under the drug plasma concentration versus time curve (AUC) after oral and intravenous administration as: In general, determinants of oral drug bioavailability include fraction of dose absorbed in the gastrointestinal tract (GIT) and fraction of dose that escapes elimination by the intestinal tract, liver, and lung
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204,205 Aberrant activation of this pathway is a well-established mechanism of resistance to targeted therapies, chemotherapy, and endocrine therapy
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To further investigate the vulnerability of the BD COs, we attempted to rescue the COs using a well-established anti-inflammatory antioxidant extract, BFE
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