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These proposed mechanisms sound compelling, but it is vital to remember that these effects are mostly observed in rodent models or cell cultures, not in humans
This is the run-in period where NNMT inhibition is being established
You read that Lee et al
By inhibiting NNMT, 5-Amino-1MQ may spare nicotinamide for NAD+ synthesis via the salvage pathway, thereby activating SIRT1 (Sirtuin 1) pathways associated with mitochondrial biogenesis, fat oxidation, and metabolic flexibility [3]
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