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Notably, the downregulation of PRR11 augments LPO and disrupts DHODH-regulated mitochondrial morphology, consequently facilitating ferroptosis and increasing TMZ chemosensitivity in GBM cells[42]

Identification of carriers and presymptomatic family members of affected individuals is achieved by polymerase-chain-reaction-based marker analysis

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The interaction between ferroptosis and inflammatory signaling pathways