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doi: 10.1007/s11102-018-0898-y

35, 36 In tumors where IR has a pathophysiologic role, anti-IGF-1R mAbs would not be expected to confer a clinical benefit if elevated insulin levels and IR activation are a result of the anti-IGF-1R therapy

Senescent endothelial cells are more susceptible to undergoing EndoMT and the proinflammatory SASP may drive the EndoMT in non-senescent endothelial cells [110]

When glutathione availability is good, hormone clearance tends to run smoothly

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