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In February 2011, it also presented the second

In the model proposed by Lee, homocysteine is shunted away from the methionine synthesis pathway and toward the GSH synthesis pathway, creating a relative deficiency of methyl donors in order to keep up with increased demand for the glutathione needed for POP conjugation ( Fig 3 )

These findings remain limited to experimental research models and do not imply clinical relevance

There are several possible reasons why vitamin E is ineffective in the treatment of human diseases