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Challenges such as solubility limitations and non-specific binding must be carefully managed

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Derived from angiotensin IV, Dihexa crosses the blood-brain barrier and promotes synaptogenesis, making it a promising candidate for treating neurodegenerative diseases such as Alzheimers and Parkinsons

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These mechanisms may accelerate the pharmacokinetic changes in the affected drug and lead to clinically relevant DDIs, particularly in drugs with a narrow therapeutic window