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Mitochondria-derived methylmalonic acid aggravates ischemia-reperfusion injury by activating reactive oxygen species-dependent ferroptosis

For this page, that source is useful as a general documentation framework, not as a peptide-specific regulatory claim

This lipid radical reacts with molecular oxygen to form a lipid peroxyl radical (LOO), which can propagate the chain reaction by abstracting hydrogen atoms from neighboring lipids, leading to the generation of more lipid radicals and peroxyl radicals [9]

Once bonds break, they don't reform

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in 2011 indicated that N-Hcy-Lys is also associated to fibrinogen, glutathione and plasminogen activator inhibitor-1 levels, and suggested the close link between N-Hcy-Lys, redox imbalance and impaired fibrinolysis in acute myocardial ischemia 10