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Even when ingested in normal amounts, copper and iron may become toxic in certain individuals with genetic predispositions that lead to hepatic overload - iron overload in hemochromatosis [11-17] or copper overload in Wilson disease [18-27]
Have undergone a comprehensive health assessment to rule out other underlying issues
The unique, upstream approach taken by Mardinoglu and team to increase endogenous glutathione synthesis and reduce fatty liver (while supporting methylation balance) based on their work using personalized genome-scale metabolic models, is worth us considering in clinical practice
Selected Research References Seiwerth S, et al
Margulies, K
