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While all GLP-1 receptor agonists effectively lower hemoglobin A1c and promote weight loss, there are meaningful differences in their magnitude of effect, titration schedules, and adverse effect profiles that influence clinical decision-making
These results indicate that the increased amount of non-12-OH FXR-antagonistic BAs consequent to the decrease in CYP8B1 expression after T3 treatment potentiates GLP-1 production by deactivating the intestinal FXR, thereby improving glucose homeostasis via the augmentation of insulin secretion
To further validate these results, we analysed a subset of genes from the Met deprivation signature, which were among the most highly enriched by GSEA, in reprogramming tissues by quantitative PCR with reverse transcription (RTqPCR)
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Insulin Insulin therapy is often required for people with type 1 diabetes or in cases where type 2 diabetes is difficult to manage with oral medications
However, long-term safety data for months of subcutaneous use at community doses does not exist
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