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IRE1-JNK pathway-mediated autophagy promotes cell survival in response to endoplasmic reticulum stress during the initial phase of hepatic steatosis

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(Rac)-1,2-Dihexadecylglycerol eliminates this artifact with non-hydrolyzable ether linkages, enabling sustained pathway modulation

Animals were exposed to standard chow or an HFD during metabolic assessment (as indicated)

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