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Disulfide bonds are relatively stable under physiological conditions but undergo selective cleavage in the tumor microenvironment (TME), minimizing premature drug release [53]

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Because many of these molecules cant be patented, pharmaceutical companies have little reason to fund those trials

"Fine mapping of the autosomal recessive polycystic kidney disease locus (PKHD1) and the genes MUT, RDS, CSNK2 beta, and GSTA1 at 6p21.1-p12"

Another study suggested that ACTL6A suppressed p21(Cip1) promoter activity to reduce p21Cip1 protein expression, which was a mechanism to maintain the aggressive phenotype of epidermal squamous cell carcinoma 14

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