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It's a complementary mechanism, not a replacement for established therapies the strongest protocols combine multiple approaches
LC and its active form, i.e., acetyl LC, can reduce systemic inflammation by reducing inflammatory factors and cytokines such as CRP, interleukin 6, and tumor necrotizing factor-alpha []
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After crossing the BBB, the GLP-1RA binds to the GLP-1R and mimics the action of the GLP-1 hormone, stimulating the intracellular accumulation of cAMP, which via the action of PKA/RGNEF4 results in neuroprotective effects (10)
Methodology This narrative review was conducted through a comprehensive literature search using databases such as PubMed, Scopus, and Web of Science
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