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Activation of IGF1R/p110/AKT/mTOR confers resistance to -specific PI3K inhibition
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S2CID 54359937
L.NykjaerC.WoodheadC
[6] Because NAD + -dependent enzymes are involved in DNA repair, protein deacetylation, stress responses, mitochondrial function, and cellular signaling, NNMT-related changes in NAD + availability should be interpreted in a pathway-specific manner rather than as a simple beneficial-or-harmful binary
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