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Penetration remains the central challenge
However, pathological modifications oxidation, glycation, citrullination, carbonylation, and enzymatic carboxyethylation of cysteine (linked to ankylosing spondylitis) can render self-proteins immunogenic, driving autoimmune disease by presenting chemically altered peptides to the immune system as foreign antigens.
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Shanghai Univ
Reduction of GSH, a vital redox modifier, affects multiple ALS associated proteins
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