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Neonatal development may also be impaired in infants breastfed by mothers who have a vitamin B12 deficiency
[Abstract] Expression of synaptophysin in mouse brains in Dihexa-treated (1.44 mg/kg and 2.88 mg/kg) APP/PS1 mice
It's frequently referenced alongside MOTS-c and NAD+ in research contexts, given the complementary nature of their respective pathway interactions - particularly how NNMT inhibition influences NAD+ availability in metabolic signalling studies
10.1016/j.bbamem.2010.10.012 186
Published research has explored its role in promoting angiogenesis (new blood vessel formation), upregulating VEGF (vascular endothelial growth factor), stimulating fibroblast migration and collagen deposition, modulating nitric oxide pathways, and protecting the gastrointestinal lining
That mechanism is plausible, but a copper complex acting in cell models does not automatically prove the same outcome in human patients
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