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Iron overload induces the separation of IRP1 from IRE-like sequence, activating NOX4 transcription, increasing lipid peroxidation accumulation, and leading to significant dysregulation of mitochondrial morphology and function of osteoblasts
doi:10.1038/s41416-021-01339-4 232
& Hanigan, M
No evidence of severe hormonal, cardiovascular, or hepatic adverse effects has been observed in available research data
Compared to solution formulas, the lyophilized powder structure is more stable over time
after five years, medical review still applies when recurrence risk or active surveillance remains relevant
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