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While androgen excess is primarily associated with reproductive pathology, physiological levels of androgens are essential for normal endometrial proliferation and differentiation through receptor-mediated mechanisms [31, 32]

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Like GSTP, GSTM1 interacts with its partner in the absence of oxidative stress, although ASK1 is also involved in heat shock response, which is likewise prevented during ASK1 sequestration

Vogt NM, Romano KA, Darst BF, et al