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Goderie T, van Wier MF, Lissenberg-Witte BI, Merkus P, Smits C, Leemans CR, Kramer SE
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The clinically normal heterozygous carriers for this disease have about half of the normal glutathione synthetase activity, as predicted, but have normal clinical phenotypes, cellular glutathione levels, and 5-oxoproline excretion (53)
Certain molds release mycotoxins, which accumulate in the body, triggering chronic inflammation and oxidative stress
qPCR analysis confirmed that Rf regulates critical genes, including ANXA2, BAZ1A, DNMT3L, and MMP9, thereby clarifying its underlying mechanism of action
"Glutamate as chemotactic fuel for diffuse glioma cells: are they glutamate suckers?"
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