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Mechanisms of human erythrocytic bioactivation of nitrite
It depends on the concentration, amount drawn, and research schedule
This mechanism represents a control of the influx of fatty acyl units into mitochondria in response to the intra-mitochondrial acetyl-CoA level, in addition to the finely regulated mechanism of CPT1 by malonyl-CoA
Heme oxygenase-1 as therapeutic target for diabetic foot ulcers
Biobank-driven genomic discovery yields new insight into atrial fibrillation biology
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