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Clinical studies indicate that GLP-1 RAs promote -cell proliferation, improve survival, and facilitate regeneration, thereby underscoring their potential for therapeutic intervention in cases of pancreatic -cell dysfunction [32]

calcium ipodate) decrease T4 production, an effect presumably mediated by release of iodine as the drug is metabolized, and also acutely inhibit peripheral T4 to T3 conversion (8)

Mouse models of diabetes and obesity demonstrate that hyperinsulinemia and insulin resistance correlate with autophagy defects, attributed to downregulation of key autophagic genes 129

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